Block NALCN, and inject stem cells into damaged organ. Normally, stem cells die off quickly when introduced into the body, but in this instance, they may be able to differentiate into the desired cell type, prompting regeneration of the damaged organ.

As for cancer, upregulate NALCN, and potentially cripple the ability of cancerous cells to migrate from the primary tumour.

If we're going to be interfering with NALCN for therapeutic effect, I wonder if there is an alternative way to maintain cellular sodium gradients, when it is either a suppressed, or upregulated state.