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Nianms t1_iz144wu wrote

Replying to [deleted] (#865,336)

Animals use lactic acid fermentation for anaerobic respiration as opposed to ethanol fermentation. So we don’t ever produce any ethanol to deal with, only lactic acid.

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uglychiccwhosthicc t1_iz1cpch wrote

It’s because of the receptors in your brain the molecules attach to. Each neurotransmitter has a different physiological effect when it’s released. Ethanol binds to GABA receptors in the brain which is an inhibitory neurotransmitter. Kinda works in a similar way to an anxiolytic (an anti-anxiety medication). They bind to the GABA receptors and that, in turn, decreases nerve transmission, which decreases neuronal excitability. One of the physiological occurrences is vasodilation

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uummbbraa t1_iz1kq47 wrote

The effects of alcohol on blood vessels can vary depending on the amount of alcohol consumed. A small amount of alcohol can cause vasodilation, which is the widening of blood vessels, while a large amount of alcohol can cause vasoconstriction, which is the narrowing of blood vessels.

The reason for this is that alcohol interacts with the walls of the blood vessels in different ways at different concentrations. At low concentrations, alcohol can cause the blood vessels to relax and become wider, which allows more blood to flow through them. This can cause the face and skin to become flushed and warm, and it can also lower blood pressure.

At high concentrations, alcohol can have the opposite effect on the blood vessels. It can cause the blood vessels to constrict and become narrower, which reduces blood flow. This can lead to decreased blood pressure, which can cause dizziness and lightheadedness. Vasoconstriction can also cause the skin to become pale and cold, and it can lead to reduced oxygen and nutrient delivery to the tissues.

The effects of alcohol on blood vessels depend on the concentration of alcohol in the blood. Small amounts of alcohol can cause vasodilation, while large amounts can cause vasoconstriction.

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OneOfUsOneOfUsGooble t1_iz1unxs wrote

"The action of alcohol on the vasculature is variable according to its concentration and the kind of blood vessel. High concentrations of alcohol constrict most blood vessels. This vasoconstriction depends on calcium ions and is inhibited by calcium channel blockers. Alcohol also acts to augment the vasoconstriction caused by catecholamines and vasopressin and inhibits endothelium-dependent vasodilation. It has been suggested that endothelin and nitric oxide are involved in alcohol-induced vasoconstriction. . . . On the other hand, low concentrations of alcohol usually dilate blood vessels. This effect also seems to be mediated by calcium ions and endothelium-derived nitric oxide. It has been shown that low doses of alcohol increase the release of nitric oxide and augment endothelium-dependent vasodilation."

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