Hi, I’m sorry but what’s wrong with what I said?

Neurons receive a large amount of information from other neurons, and the dendrites' role is to integrate and process this information. The information is then filtered, and only the relevant information is transmitted along the axon to other neurons. The more dendrites you have, the more information you can receive.

The filtering of information occurs through a process known as synaptic plasticity, where the strength of the synapses between neurons can be adjusted based on the input received. Inhibitory neurotransmitters can also play a role in filtering information, as they can decrease the likelihood that an action potential will be generated in the neuron.

Let’s say you have nerves for sensing pain on the tip of your index. They all need to cross the same path to make their way to the spinal cord (dorsal horn). So the body has lots of these pain sensing nerves on the tip of your fingers that connect to a larger neuron that fires a strong and incredibly fast signal to the dorsal horn. Then we get a signal that fires back from spinal cord level to your finger to pull it back and then the process gets processed in the brain.

Fingers could be the same length. Women tend to have same sized ring and index fingers, meanwhile the ring finger tends to be longer than the index finger in male population (especially men exposed to higher concentrations of testosterone during fetal period.

Some studies suggest that larger fingers may give you an advantage in some sports, but it’s not fully understood whether fingersize really matters evolution wise.

Don’t hesitate to correct me if I am wrong.

I don’t understand what you mean. Corticosteroids can cause adrenal insufficiency in the long term. Overdosage of Flonase can cause it.

People should be wary of what they intake and should seek professional help on whether they should be taking such medication for such prolonged time. OP was curious and I gave him the answer.

However they interpret my answer is up to them.

Edit: also if you search it on google you’ll see that the is indeed happened before from taking too much of Flonase.

Yeah it’s definitely very unlikely, but definitely something to consider and to mention.

Plus OP just asked a question and I gave them the most logical answer, whether anyone actually got it is something I don’t know.

>Flonase can lead to a condition called rhinitis medicamentosa, or rebound congestion.

It varies from person to person of course, and how well their body is able to handle the drug metabolism.

For childeren, prolonged intake of 100 to 200 ug daily may indeed increase the risk, while for adults the dosis might be up to 500 ug.

Of course people with already existing adrenal gland problems would handle it worse compared to normal people and they would see the side effects much faster.

Yes. Cells may become multinucleated cells if something goes wrong during the cell division/fusion which is possible when cancerous cells divide way too often unregulatedly.

It’s more complex than it looks like. The body randomly creates many different antibodies against many different type of antigens. The one that’s able to bind on the antigen on the virus is the one that will start reproducing and creating millions of itself if necessary to fight the specific virus.

The two antibodies of the two different persons will be most likely be different. One of them will probably have a higher affinity towards the virus compared to the other person which will mean that that person may also fight the virus faster.

I can’t go into much more detail since I still haven’t been taught much about this topic yet.

Source: med student

Like i said in my comment above, leptin hormone starts spiking during the age of 10 which releases the inhibition on KDNy-neurons and thus we get release of Kiss peptides, thus GnRH starts getting released and we get FSH and LH (thus Testosterone)

Check my other comment in this thread for more detail

Yeah I mean lots of people know the basics about these hormones, but relatively few know the details. Glad I could help.

Haha it's crazy how the body is made. This hormone produces this which produces this which produces this x100

Well if you want more detail, while the fetus is in the placenta (a place where lots of estrogen is located), we get inhibition of the KDNy neurons. (see above)

After birth, the high [] of estrogen that came from the mother goes away, so the KDNy neurons start releasing Kiss peptide => stimulation of the GnRH-neuron => LH and FSH release => testosteron + estrogen production

LH stimulates the production of progesteron and other androgens which leads to a positive effect on this cycle.

After 3 months the GABA hormone concentration starts getting high enough to block the KDNy neuron activity => the LH and FSH production (and testosteron as well) stops from the age of 6months until the age of 10 years old, where leptine starts getting produced as well and we get a more positive feedback on the testosteron production.

Edit: something like this

I also wanna say many other hormones play a role, but to keep it simple I only mentioned the important ones.

GNRH release is regulated by KNDY-neurons (Kisspeptide-neurokinine B -dynorphine)

The KDNY neurons get stimulated by progesteron, we get release of Kisspeptide which stimulates the GNRH neuron and releases GNRH

GnRH then binds on the gonadotropic cells which causes the release of FSH and LHLH and FSH stimulate the production of testosteron and estrogen then inhibits the KDNy-neurons (negative feedback loop) so we don't get too much of GNRH release

Edit: I don't exactly know what causes this release in the first place, but studies have shown that leptine, a hormone secreted by fat cells in the human body, stimulate the KNDY-neurones which could lead to the start of the GnRH release.

During puberty (10-12yo) the child starts having lots of lipid cells which thus means more leptin secretion. This also means the "fatter" childeren get a faster puberty. It's a process called the gonadarche.

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source: med student